, Federico II University, Naples, ItalyGuido Iaccarino & Bruno Trimarco AuthorsSearch for Guido Iaccarino in:PubMed • Google Scholar Search for Bruno Trimarco in:PubMed • Google Scholar Contributions GI
. Ipertensione e menopausa Bruno Trimarco Che relazione c’è tra ipertensione arteriosa e menopausa? L’ipertensione essenziale rappresenta il maggior determinante di morbilità e mortalità cardiovascolare (CV ... dell’ipertensione in menopausa. Il ruolo dell’automisurazione domiciliare Bruno Trimarco Cosa s’intende per automisurazione della pressione arteriosa? L’automisurazione, o misurazione della pressione arteriosa
In Anderson–Fabry disease (AFD) the impact of left ventricular (LV) function on cardiac outcome is unknown. Noninvasive LV pressure–strain loop analysis is a new echocardiographic method to estimate myocardial work (MW). We aimed to evaluate whether LV function was associated with outcome and whether MW had a prognostic value in AFD. Ninety-six AFD patients (41.8 ± 14.7 years, 43...
, Federico II University Hospital, Naples, ItalyCiro Santoro, Regina Sorrentino, Roberta Esposito, Maria Lembo, Valentina Capone, Francesco Rozza, Massimo Romano, Bruno Trimarco & Maurizio Galderisi ... • Google Scholar Search for Valentina Capone in:PubMed • Google Scholar Search for Francesco Rozza in:PubMed • Google Scholar Search for Massimo Romano in:PubMed • Google Scholar Search for Bruno Trimarco
The discovery of the molecular mechanisms involved in the cardiac responses to anticancer drugs represents the current goal of cardio-oncology research. The oxidative stress has a pivotal role in cardiotoxic responses, affecting the function of all types of cardiac cells, and their functional crosstalks. Generally, cardiomyocytes are the main target of research studies on...
The discovery of the molecular mechanisms involved in the cardiac responses to anticancer drugs represents the current goal of cardio-oncology research. The oxidative stress has a pivotal role in cardiotoxic responses, affecting the function of all types of cardiac cells, and their functional crosstalks. Generally, cardiomyocytes are the main target of research studies on...
The discovery of the molecular mechanisms involved in the cardiac responses to anticancer drugs represents the current goal of cardio-oncology research. The oxidative stress has a pivotal role in cardiotoxic responses, affecting the function of all types of cardiac cells, and their functional crosstalks. Generally, cardiomyocytes are the main target of research studies on...
The discovery of the molecular mechanisms involved in the cardiac responses to anticancer drugs represents the current goal of cardio-oncology research. The oxidative stress has a pivotal role in cardiotoxic responses, affecting the function of all types of cardiac cells, and their functional crosstalks. Generally, cardiomyocytes are the main target of research studies on...
The discovery of the molecular mechanisms involved in the cardiac responses to anticancer drugs represents the current goal of cardio-oncology research. The oxidative stress has a pivotal role in cardiotoxic responses, affecting the function of all types of cardiac cells, and their functional crosstalks. Generally, cardiomyocytes are the main target of research studies on...
Regina Sorrentino in:PubMed • Google Scholar Search for Maria Lembo in:PubMed • Google Scholar Search for Covadonga Fernandez-Golfin in:PubMed • Google Scholar Search for Bruno Trimarco in:PubMed
Cardiovascular disease and heart failure still collect the largest toll of death in western societies and around the world. A growing number of molecular mechanisms represent possible targets for novel therapeutic strategies, in order to counteract the metabolic and structural changes observed in the failing heart. GRK2 is one of such targets for which experimental and clinical...
Nella Prevete,2,3 Antonella Fiordelisi,1 Michele Ciccarelli,4 Bruno Trimarco,1 Nicola De Luca,1 and Guido Iaccarino1 1Dept. of Advanced Biomedical Sciences, Federico II University, Napoli, Italy 2Dept
Nella Prevete,2,3 Antonella Fiordelisi,1 Michele Ciccarelli,4 Bruno Trimarco,1 Nicola De Luca,1 and Guido Iaccarino1 1Dept. of Advanced Biomedical Sciences, Federico II University, Napoli, Italy 2Dept
Nella Prevete,2,3 Antonella Fiordelisi,1 Michele Ciccarelli,4 Bruno Trimarco,1 Nicola De Luca,1 and Guido Iaccarino1 1Dept. of Advanced Biomedical Sciences, Federico II University, Napoli, Italy 2Dept
MAFG (v-Maf avian musculoaponeurotic fibrosarcoma oncogene homolog G) is a bZIP-type transcriptional regulator that belongs to the small MAF (sMAFs) protein family. By interacting with other bZIP transcription factors, sMAFs can form homo- and heterodimers governing either repressive or activating transcriptional functions. As heterodimeric partner of Nrf2, MAFG positively...
MAFG (v-Maf avian musculoaponeurotic fibrosarcoma oncogene homolog G) is a bZIP-type transcriptional regulator that belongs to the small MAF (sMAFs) protein family. By interacting with other bZIP transcription factors, sMAFs can form homo- and heterodimers governing either repressive or activating transcriptional functions. As heterodimeric partner of Nrf2, MAFG positively...
MAFG (v-Maf avian musculoaponeurotic fibrosarcoma oncogene homolog G) is a bZIP-type transcriptional regulator that belongs to the small MAF (sMAFs) protein family. By interacting with other bZIP transcription factors, sMAFs can form homo- and heterodimers governing either repressive or activating transcriptional functions. As heterodimeric partner of Nrf2, MAFG positively...
