. Reprint requests: K. Lance Gould, MD, Weatherhead PET Center for Preventing and Reversing Atherosclerosis, McGovern Medical School, University of Texas Health Science Center at Houston , 6431 Fannin St
institutional research support from Volcano Corporation (for study NCT02328820) and St Jude Medical (for study NCT02184117), companies that make intracoronary pressure and flow sensors. Reprint requests: K. Lance ... Gould, MD, Division of Cardiology, Department of Medicine, Weatherhead PET Center for Preventing and Reversing Atherosclerosis, University of Texas Medical School at Houston and Memorial Hermann Hospital
Background Heterogeneity of resting perfusion may be due in part to up-regulation of coronary vasoconstriction via endothelin (ET) type A receptors, as homogeneity increases during subsequent vasodilatory hyperemia. Therefore, we conducted a mechanistic study using an ET receptor antagonist to determine if it could alter the homogeneity of myocardial perfusion. Methods Included...
Nils P. Johnson MS K. Lance Gould MD Why do we model myocardial perfusion? The article by Alessio et al1 in this issue offers a timely opportunity to consider two major, competing reasons. In turn
0 Nils P. Johnson , MD , MS K. Lance Gould, MD Weatherhead PET Center for Preventing and Reversing Atherosclerosis, Division of Cardiology, Department of Medicine, University of Texas Medical School
Nils P. Johnson Stefano Sdringola K. Lance Gould MD Background. Quantitative myocardial PET perfusion imaging requires partial volume corrections. Methods. Patients underwent ECG-gated, rest