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Burkholderia pseudomallei natural competency and DNA catabolism: Identification and characterization of relevant genes from a constructed fosmid library

, P20GM103516, U54 AI065359. Author Contributions Conceptualization: Tung T. Hoang. Formal analysis: Michael H. Norris, Yun Heacock-Kang. Methodology: Michael H. Norris, Yun Heacock-Kang, Jan Zarzycki-Siek ... , Andrew P. Bluhm, Ian A. McMillan. Resources: Herbert P. Schweizer. 15 / 17 Supervision: Tung T. Hoang. Validation: Michael H. Norris, Yun Heacock-Kang, Jan Zarzycki-Siek, Andrew P. Bluhm, Ian A

Development of Immunoassays for Burkholderia pseudomallei Typical and Atypical Lipopolysaccharide Strain Typing

. Michael H. Norris and Apichai Tuanyok, Department of Infectious Diseases and Pathology, University of Florida, Gainesville, FL, E-mails: and . Paul J. Brett and Mary N. Burtnick, Department of Microbiology

Blocking Phosphatidylcholine Utilization in Pseudomonas aeruginosa, via Mutagenesis of Fatty Acid, Glycerol and Choline Degradation Pathways, Confirms the Importance of This Nutrient Source In Vivo

Pseudomonas aeruginosa can grow to very high-cell-density (HCD) during infection of the cystic fibrosis (CF) lung. Phosphatidylcholine (PC), the major component of lung surfactant, has been hypothesized to support HCD growth of P. aeruginosa in vivo. The phosphorylcholine headgroup, a glycerol molecule, and two long-chain fatty acids (FAs) are released by enzymatic cleavage of PC...

Elucidating the Pseudomonas aeruginosa Fatty Acid Degradation Pathway: Identification of Additional Fatty Acyl-CoA Synthetase Homologues

The fatty acid (FA) degradation pathway of Pseudomonas aeruginosa, an opportunistic pathogen, was recently shown to be involved in nutrient acquisition during BALB/c mouse lung infection model. The source of FA in the lung is believed to be phosphatidylcholine, the major component of lung surfactant. Previous research indicated that P. aeruginosa has more than two fatty acyl-CoA...

Multiple FadD Acyl-CoA Synthetases Contribute to Differential Fatty Acid Degradation and Virulence in Pseudomonas aeruginosa

A close interconnection between nutrient metabolism and virulence factor expression contributes to the pathophysiology of Pseudomonas aeruginosa as a successful pathogen. P. aeruginosa fatty acid (FA) degradation is complicated with multiple acyl-CoA synthetase homologs (FadDs) expressed in vivo in lung tissue during cystic fibrosis infections. The promoters of two genetically...