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13 papers found.
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Development of novel predictive miRNA/target gene pathways for colorectal cancer distance metastasis to the liver using a bioinformatic approach

(DEGs) between GSE49355, GSE41258 and GSE81558. (XLSX) 15 / 20 Author Contributions Conceptualization: Po-Li Wei, Yu-Jia Chang. Data curation: Precious Takondwa Makondi, Po-Li Wei, Chien-Yu Huang, Yu ... -Jia Chang. Formal analysis: Precious Takondwa Makondi, Po-Li Wei, Yu-Jia Chang. Funding acquisition: Yu-Jia Chang. Investigation: Precious Takondwa Makondi, Yu-Jia Chang. Methodology: Precious

Propyl gallate inhibits hepatocellular carcinoma cell growth through the induction of ROS and the activation of autophagy

by a grant from Taipei Medical University and Taipei Medical University Hospital (106TMU-TMUH-07). Author Contributions Conceptualization: Po-Li Wei, Yu-Jia Chang. Data curation: Po-Li Wei, Chien-Yu ... Huang, Yu-Jia Chang. Formal analysis: Chien-Yu Huang, Yu-Jia Chang. Investigation: Po-Li Wei, Chien-Yu Huang, Yu-Jia Chang. Methodology: Yu-Jia Chang. Project administration: Yu-Jia Chang. Resources: Po

Bromelain inhibits the ability of colorectal cancer cells to proliferate via activation of ROS production and autophagy

, Taipei Medical University (105TMU-SHH-19). Author Contributions Conceptualization: Po-Li Wei, Yu-Jia Chang. Huang, Yu-Jia Chang. Formal analysis: Po-Li Wei, Yu-Jia Chang. Funding acquisition: Chien-Yu ... Huang. Data curation: Tung-Cheng Chang, Po-Li Wei, Precious Takondwa Makondi, Chien-Yu Investigation: Tung-Cheng Chang, Po-Li Wei, Wei-Ting Chen, Chien-Yu Huang. Methodology: Precious Takondwa Makondi

Prediction of novel target genes and pathways involved in bevacizumab-resistant colorectal cancer

findings and determine their clinical applicability. Supporting information S1 Table. Down-regulated bevacizumab-resistant genes. (DOCX) 12 / 18 Conceptualization: Chien-Yu Huang, Chi-Ming Chu, Po-Li Wei ... . Data curation: Precious Takondwa Makondi, Chia-Hwa Lee, Yu-Jia Chang. Investigation: Precious Takondwa Makondi, Chi-Ming Chu, Po-Li Wei. S2 Table. Up-regulated genes. (DOCX) Author Contributions Formal

Prediction of novel target genes and pathways involved in irinotecan-resistant colorectal cancer

Background Acquired drug resistance to the chemotherapeutic drug irinotecan (the active metabolite of which is SN-38) is one of the significant obstacles in the treatment of advanced colorectal cancer (CRC). The molecular mechanism or targets mediating irinotecan resistance are still unclear. It is urgent to find the irinotecan response biomarkers to improve CRC patients’ therapy...

Comparison of clinical outcome between laparoscopic and open right hemicolectomy: a nationwide study

Background This study aimed to compare clinical outcome between laparoscopic and open right hemicolectomy. Methods The data were sourced from Taiwan’s National Health Insurance Research Database. This study included 14,320 and 1313 patients who underwent open and laparoscopic right hemicolectomies, respectively. The study outcome included “intensive care unit (ICU) admission...

Thrombomodulin Influences the Survival of Patients with Non-Metastatic Colorectal Cancer through Epithelial-To-Mesenchymal Transition (EMT)

Background Treatment resistance and metastasis are the major causes of death among patients with colorectal cancer (CRC). Approximately 20% of surgically treated patients ultimately develop metastases during the follow-up period. Currently, the TNM system is the only available prognostic test. Therefore, the identification of new markers for CRC remains important. Thrombomodulin...

A Novel Microtubule-Disrupting Agent Induces Endoplasmic Reticular Stress-Mediated Cell Death in Human Hepatocellular Carcinoma Cells

Here, we present evidence of a novel microtubule-disrupting agent, N-deacetyl-N-(chromone-2-carbonyl)-thiocolchicine (TCD), exhibiting potent antitumor activity (with IC50 values in the nanomolar range) against hepatocellular carcinoma cell lines. Cell cycle analysis revealed that TCD induced G2/M cell-cycle arrest in a dose- and time-dependent manner in both Hep-J5 and Mahlavu...

Volume-Outcome Relation for Acute Appendicitis: Evidence from a Nationwide Population-Based Study

Background Although procedures like appendectomy have been studied extensively, the relative importance of each surgeon's surgical volume-to-ruptured appendicitis has not been explored. The purpose of this study was to investigate the rate of ruptured appendicitis by surgeon-volume groups as a measure of quality of care for appendicitis by using a nationwide population-based...

Outcome Comparison between Laparoscopic and Open Appendectomy: Evidence from a Nationwide Population-Based Study

Background Mounting evidence supports the use of laparoscopic techniques for the treatment of simple appendicitis. However, most of the advantages of these techniques are of limited clinical relevance. This study compares the treatment outcomes of laparoscopic appendectomies and open appendectomies performed in Taiwan. Methods This study uses data from the 2007 to 2009 Taiwan...

Slow Freezing Coupled Static Magnetic Field Exposure Enhances Cryopreservative Efficiency—A Study on Human Erythrocytes

The aim of this study was to assess the cryoprotective effect of static magnetic fields (SMFs) on human erythrocytes during the slow cooling procedure. Human erythrocytes suspended in 20% glycerol were slowly frozen with a 0.4-T or 0.8-T SMF and then moved to a −80°C freezer for 24 hr. The changes in survival rate, morphology, and metabolites of the thawed erythrocytes were...

Erratum to: Survivin-mediated Therapeutic Efficacy of Gemcitabine through Glucose-regulated Protein 78 in Hepatocellular Carcinoma

Kuo Li-Tzu Li Hou-Yu Su Phui-Ly Liew Feng-Yen Lin Po-Li Wei Der-Zen Liu Yu-Jia Chang - FIG. 1 The percentages of the viable HepJ5 and Mahlavu cells after treated with gemcitabine. a The survivin-KD

Overexpression and Activation of the α9-Nicotinic Receptor During Tumorigenesis in Human Breast Epithelial Cells

Background Large epidemiological cohort studies in the United States have indicated that active and passive smoking are associated with increased breast cancer risk. However, there was no direct evidence of an effect of tobacco carcinogens on the cellular molecules involved in breast tumorigenesis.