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8 papers found.
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Cell cycle checkpoint in cancer: a therapeutically targetable double-edged sword

Major currently used anticancer therapeutics either directly damage DNA or target and upset basic cell division mechanisms like DNA replication and chromosome segregation. These insults elicit activation of cell cycle checkpoints, safeguard mechanisms that cells implement to correctly complete cell cycle phases, repair damage or eventually commit suicide in case damage is...

Cell cycle checkpoint in cancer: a therapeutically targetable double-edged sword

Major currently used anticancer therapeutics either directly damage DNA or target and upset basic cell division mechanisms like DNA replication and chromosome segregation. These insults elicit activation of cell cycle checkpoints, safeguard mechanisms that cells implement to correctly complete cell cycle phases, repair damage or eventually commit suicide in case damage is...

Fcp1 phosphatase controls Greatwall kinase to promote PP2A-B55 activation and mitotic progression

://dx.doi.org/10.7554/eLife.10399 Cite as: eLife 2015;10.7554/eLife.10399 Rosa Della Monica Roberta Visconti Nando Cervone Angela Flavia Serpico Domenico Grieco - Stay current on the latest in life science and

Fcp1-dependent dephosphorylation is required for M-phase-promoting factor inactivation at mitosis exit

Biotechnological Sciences, DBPCM, University of Naples 'Federico II', via S. Pansini 5, 80131 Naples, Italy.Luca Palazzo, Rosa Della Monica & Domenico Grieco AuthorsSearch for Roberta Visconti in:Nature Research

Janus kinases and signal transducers and activators of transcription: their roles in cytokine signaling, development and immunoregulation

Frucht, Roberta Visconti and John J O’Shea (Lymphocyte Cell Biology Section, Arthritis and Rheumatism Branch, National Institute of Arthritis, Musculoskeletal and Skin Diseases, National Institutes of

Cloning and molecular characterization of a novel gene strongly induced by the adenovirus E1A gene in rat thyroid cells

Expression of the adenovirus E1A gene in the rat thyroid differentiated cell line PC Cl 3 induces thyrotropin-independent cell growth and impairs differentiation. However, the malignant phenotype is achieved only when the PC E1A cells are infected with other murine retroviruses carrying the v-abl, v-raf or polyoma middle-T genes. To determine through which genes E1A affects...

Expression of the neoplastic phenotype by human thyroid carcinoma cell lines requires NFκB p65 protein expression

We have investigated the role of the NFκB complex in the process of thyroid carcinogenesis by analysing thyroid carcinoma cell lines. A significant increase in p65 NFκB mRNA and protein expression, compared to normal thyroid cultures or tissue, was found in all of the cancer cell lines. Conversely, only a modest increase in the p50 NFκB mRNA and protein was found in most, but not...