Author Correction: The EphA2 receptor is activated through induction of distinct, ligand-dependent oligomeric structures

DOI: 10.1038/s42003-018-0044-4 OPEN 1234567890():,; Author Correction: The EphA2 receptor is activated through induction of distinct, ligand-dependent oligomeric structures Deo R. Singh1,2, Pranjali Kanvinde1,2, Christopher King1,3, Elena B. Pasquale4,5 & Kalina Hristova1,2,3 Correction to: Communications Biology https://doi.org/10.1038/s42003-018-0017-7, published online 22 February 2018. The original published version of the paper contained labelling errors in Figures 1 and 5. The x-axis of Figure 1e was incorrectly labelled as “Receptors . μm2” instead of “Receptors . μm−2”. The figure key in Figure 5b incorrectly labelled the blue circles as “Wildtype” and the purple circles as “+m-ephrinA1”. The correct labels are blue: “Wild-type+m-ephrinA1” and purple: “Wild-type, no ligand.” The original version also contained a typesetting error in Table 1. The values in the second row, rightmost column, were not all set in bold text as required. The errors do not impact the conclusions in the main text of the paper. These errors have been corrected in the PDF and HTML versions of the paper. Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/. © The Author(s) 2018 1 Department of Materials Science and Engineering, Johns Hopkins University, 3400 Charles Street, Baltimore, MD 21218, USA. 2 Institute of NanoBioTechnology, Johns Hopkins University, 3400 Charles Street, Baltimore, MD 21218, USA. 3 Program in Molecular Biophysics, Johns Hopkins University, 3400 Charles Street, Baltimore, MD 21218, USA. 4 Sanford Burnham Prebys Medical Discovery Institute, 10901 North Torrey Road, La Jolla, CA 92037, USA. 5 Pathology Department, University of California San Diego, La Jolla, CA 92093, USA. Correspondence and requests for materials should be addressed to K.H. (email: ) COMMUNICATIONS BIOLOGY | (2018)1:40 | DOI: 10.1038/s42003-018-0044-4 | www.nature.com/commsbio 1  (...truncated)